Ventricular Tachycardia

This is part of: Ventricular Arrhythmias

Ventricular Tachycardia (VT or V-tach)
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Atrial rate	60-100 bpm
Ventricular rate	110-250 bpm
Regularity	regular
Origin	ventricles
P-wave	AV-dissociation
Effect of adenosine	no rate reduction (can accelerate)
Example ECG: An example of a polymorphic ventricular tachycardia
Example ECG2: Ventricular tachycardia on a 12 lead recording Ventricular tachycardia of 140 bpm with a left bundle branch block and a left axis.

Ventricular tachycardia is defined as a sequence of three or more ventricular beats. The frequency must by higher than 100 bpm, mostly it is 110-250 bpm. Ventricular tachycardias often origin around old scar tissue in the heart, e.g. after myocardial infarction. Also electrolyte disturbances and ischemia can cause ventricular tachycardias. The cardiac output is often strongly reduced during VT resulting in hypotension and loss of conciousness. VT is a medical emergency as it can deteriorate into Ventricular fibrillation and thus mechanical cardiac arrest. Although ventricular tachycardia is often associated with cardiac disease, short non-sustained VTs during exercise in otherwise healthy individuals, are not necesarrily associated with a worse prognosis [1].

Ventricular tachycardia can be catechorized as follows:

• Non-sustained VT: three or more ventricular beats with a maximal duration of 30 seconds.
• Sustained VT: a VT of more than 30 seconds duration (or less if treated by electrocardioversion within 30 seconds).
• Monomorphic VT: all ventricular beats have the same configuration.
• Polymorphic VT: the ventricular beats have a changing configuration. The RR interval is 180-600 ms (comparable to a heart rate of 100-333 bpm).
• Biphasic VT: a ventricular tachycardia with a QRS complex that alternates from beat to beat. Associated with digoxin intoxication and long QT syndrome.

Ventricular tachycardia can be difficult to diagnose when confronted with a wide complex tachycardia. A seperate chapter deals with ECG algorithms to analyze wide complex tachycardias.

Localisation of the origin of a ventricular tachycardia

The localisation of the origin (or exit site) of a ventricular tachycardia can be helpful in understanding the cause of the VT and is very helpful when planning an ablation procedure to treat a ventricular tachycardia. Localisation of the origin of a ventricular tachycardia is discussed in a seperate chapter.

Examples

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  Ventricular tachycardia of 140 bpm with a left bundle branch block pattern and left heart axis.

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  Ventricular tachycardia of 250 bpm with a right bundle branch block pattern and right heart axis.

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  Ventricular tachycardia of 150 bpm with a right bundle branch block pattern and right heart axis. Mind the 5th and 6th complex from the right side. These are fusion complexes. Furthermore this ECG shows baseline drift, which is a technical artefact

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  Ventricular tachycardia of 145 bpm with a right bundle branch block pattern and left heart axis.

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  A non sustained VT of five beats duration

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  An example of idiopathic ventricular tachycardia (Belhassen VT)

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  A biphasic VT in a patient with long QT syndrome

References

1. Marine JE, Shetty V, Chow GV, Wright JG, Gerstenblith G, Najjar SS, Lakatta EG, and Fleg JL. Prevalence and prognostic significance of exercise-induced nonsustained ventricular tachycardia in asymptomatic volunteers: BLSA (Baltimore Longitudinal Study of Aging). J Am Coll Cardiol. 2013 Aug 13;62(7):595-600. DOI:10.1016/j.jacc.2013.05.026 |
2. Segal OR, Chow AW, Wong T, Trevisi N, Lowe MD, Davies DW, Della Bella P, Packer DL, and Peters NS. A novel algorithm for determining endocardial VT exit site from 12-lead surface ECG characteristics in human, infarct-related ventricular tachycardia. J Cardiovasc Electrophysiol. 2007 Feb;18(2):161-8. DOI:10.1111/j.1540-8167.2007.00721.x |