Clinical Disorders: Difference between revisions
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{{authors| | {{authors| | ||
|mainauthor= [[user:Vdbilt|I.A.C. van der Bilt, MD]] | |mainauthor= [[user:Vdbilt|I.A.C. van der Bilt, MD]] | ||
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*prominent u-waves. | *prominent u-waves. | ||
These abnormalites are frequently seen after [[w:Subarachnoid_hemorrhage|subarachnoid_hemorrhage (SAH)]] (if measured serially, almost every SAH patients has at least one abnormal ECG.), but also in [[w:Subdural_haematoma|subdural haematoma]], ischemic [[w:Cerebrovascular_accident|CVA]]'s, [[w:Brain_tumor|brain Tumors]], [[w:Guillain-Barre|Guillain | These abnormalites are frequently seen after [[w:Subarachnoid_hemorrhage|subarachnoid_hemorrhage (SAH)]] (if measured serially, almost every SAH patients has at least one abnormal ECG.), but also in [[w:Subdural_haematoma|subdural haematoma]], ischemic [[w:Cerebrovascular_accident|CVA]]'s, [[w:Brain_tumor|brain Tumors]], [[w:Guillain-Barre|Guillain Barré]], [[w:Epilepsy|epilepsy]] and [[w:Migraine|migraine]]. The ECG changes are generally reversible and have linited prognostic value. However, the ECG changes can be accompanied with myocardial damage and echocardiographic changes. The cause of the ECG changes is not yet cl;ear. The most common hypothesis is that of a neurotramittor "catecholaminestorm" caused by sympathtic stimulation. | ||
==Cardiac contusion== | ==Cardiac contusion== | ||
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#Rodger pmid=11018210 | #Rodger pmid=11018210 | ||
#Ferrari pmid=9118684 | #Ferrari pmid=9118684 | ||
#Aschenbrenner Aschenbrenner R, Bodechtel G, '' | #Aschenbrenner Aschenbrenner R, Bodechtel G, ''Ãber Ekg.-Veränderungen bei Hirntumorkranken''. Journal of Molecular Medicine, 17, 9, 2/1/1938, Pages 298-302, http://dx.doi.org/10.1007/BF01778563 | ||
#Maron pmid=14681516 | #Maron pmid=14681516 | ||
#hypoth pmid=2738372 | #hypoth pmid=2738372 | ||
</biblio> | </biblio> | ||