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| Culprit lesion: '''RCX'''
| | {{Case| |
| | |previouspage= MI 4 |
| | |previousname= MI 4 |
| | |nextpage=MI 6 |
| | |nextname=MI 6 |
| | }} |
| | '''Where is this myocardial infarction located?''' |
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| # sinus rhythm
| | [[Image:ami0005.jpg|700px|thumb|left|ECG MI 5]] |
| # about 60/min
| | {{clr}} |
| # normal conduction
| | [[Answer MI 5|Answer]] |
| # intermediate axis
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| # normal p wave morphology
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| # No pathologic Q or LVH. Tall R in V2, V3.
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| # ST depression in V2, V3. Also depression in III and AVF. Some elevation in I and AVL.
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| * Conclusion: '''Postero-lateral MI caused by an RCX occlusion.'''
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| Note! The high frequency vibration that is most clearly seen in lead AVR (with a frequency of > 300/min) is an artefact and not a suprvaventricular tachycardia. In SVT, there would be no P waves.
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| It is quite unusual that lead III shows depression in a RCX infarction. Apparently the inferior part is not much affected by this infarction.
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