Repolarization (ST-T,U) Abnormalities: Difference between revisions

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Repolarization can be influenced by many factors, including electrolyte shifts, ischemia, structural heart disease (cardiomyopathy) and (recent) arrhythmias. Although T/U wave abnormalities are rarely specific for one disease, it can be useful to know which conditions can change repolarization.
Repolarization can be influenced by many factors, including electrolyte shifts, ischemia, structural heart disease (cardiomyopathy) and (recent) arrhythmias. Although T/U wave abnormalities are rarely specific for one disease, it can be useful to know which conditions can change repolarization.


*Early repolarization (normal variant)
*Early repolarization (normal variant)[[File:early_repol.svg|thumb|300px]]
*Juvenile T waves (normal variant)
*Juvenile T waves (normal variant)
*Nonspecific abnormality, ST segment and/or T wave
*Nonspecific abnormality, ST segment and/or T wave
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*Prominent U waves
*Prominent U waves
*Cardiac Memory|Cardiac Memory
*Cardiac Memory|Cardiac Memory
'''Early repolarization''' is a normal variant of the ST segment, seen in 2-5% of patients, especially young men. Early repolarization is characterized by elevation of the J point and the beginning of the ST segment as well as elevation of the ST segment itself<cite>Wellens</cite>. The ST segment may be concave up (cup-like) or concave (dome-like). These findings are most often present in the middle chest leads V2-V5.
Recently a different form of early repolarization has been associated with idiopathic ventricular fibrillation. This form is most often seen in lead II and consists of a 'hump' in the tail of the QRS complex, without ST elevation.
==References==
<biblio>
#Wellens pmid=18463384
#Tikkanen pmid=19917913
</biblio>

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