Anterior MI: Difference between revisions
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Proximal or distal occlusion of the LAD can be differentiated when looking at the ST elevation V1-V3 <cite>Zimetbaum</cite> | Proximal or distal occlusion of the LAD can be differentiated when looking at the ST elevation V1-V3 <cite>Zimetbaum</cite> | ||
;Characteristics of proximal LAD occlusion | ;Characteristics of proximal LAD occlusion | ||
*ST-segment elevation in V1 (>2.5 mm) or [[RBBB]] with a pathologic [[Pathologic Q Waves|Q wave]] or both | *ST-segment elevation in V1 (>2.5 mm) or [[RBBB]] with a pathologic [[Pathologic Q Waves|Q wave]] or both (sens 12%, spec 100%) | ||
*ST-segment depression (>1 mm) in II, III and aVF | *ST-segment depression (>1 mm) in II, III and aVF (sens 34%, spec 98%) | ||
;Characteristics of distal LAD occlusion | ;Characteristics of distal LAD occlusion | ||
*Little ST-segment depression (<= 1 mm) or even elevation in II, III, and aVF | *Little ST-segment depression (<= 1 mm) or even elevation in II, III, and aVF (sens 66%, spec 73%) | ||
Another way to look at this is by assessing the axix of the ST vector. If it points upwards (with ST depression in II, III, and AVF) the proximal LAD is occluded. If it points downwards (with little ST depression or even elevation in II, III, and AVF) the distal LAD is occluded. An ECG that does not show any ST depression sugggests an occlusion after the origin of the first diagonal branch. | Another way to look at this is by assessing the axix of the ST vector. If it points upwards (with ST depression in II, III, and AVF) the proximal LAD is occluded. If it points downwards (with little ST depression or even elevation in II, III, and AVF) the distal LAD is occluded. An ECG that does not show any ST depression sugggests an occlusion after the origin of the first diagonal branch. | ||
Revision as of 08:58, 25 July 2007
| This is part of: Myocardial Infarction |
ECG-characteristics:Wung
ST-elevation in leads V1-V6, I and aVL. Maximum elevation in V3, maximal depression in III later: pathological Q-wave in the precordial leads V2 to V4-V5.
Anterior MI can involve the anterior part of the heart and a part of the ventricular septum. Is supplied by blood by the LAD. Can lead to a cardiac aneurysm if not treated timely.
Proximal or distal occlusion of the LAD can be differentiated when looking at the ST elevation V1-V3 Zimetbaum
- Characteristics of proximal LAD occlusion
- ST-segment elevation in V1 (>2.5 mm) or RBBB with a pathologic Q wave or both (sens 12%, spec 100%)
- ST-segment depression (>1 mm) in II, III and aVF (sens 34%, spec 98%)
- Characteristics of distal LAD occlusion
- Little ST-segment depression (<= 1 mm) or even elevation in II, III, and aVF (sens 66%, spec 73%)
Another way to look at this is by assessing the axix of the ST vector. If it points upwards (with ST depression in II, III, and AVF) the proximal LAD is occluded. If it points downwards (with little ST depression or even elevation in II, III, and AVF) the distal LAD is occluded. An ECG that does not show any ST depression sugggests an occlusion after the origin of the first diagonal branch.
Examples
-
A typical example of an acute anterior wall infarction. ST elevation in leads I, AVL and V2-V5. Reciprocal depressions in the inferior leads (II,III,AVF) -
Acute MI with proximal LAD occlusion -
Large acute MI with LAD occlusion -
Acute MI with LAD occlusion -
A 2 weeks old anterior infarction with Q waves in V2-V4 and persisting ST elevation, a sign of formation of a cardiac aneurysm.
References
<biblio>
- Wung pmid=16777513
- Zimetbaum pmid=12621138
</biblio>