Anterior MI: Difference between revisions

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Image:AMI_anterior.png|A typical example of an acute anterior wall infarction. ST elevation in leads I, AVL and V2-V5. Reciprocal depressions in the inferior leads (II,III,AVF)
Image:AMI_anterior.png|A typical example of an acute anterior wall infarction. ST elevation in leads I, AVL and V2-V5. Reciprocal depressions in the inferior leads (II,III,AVF)
Image:Ami0003.jpg|Acute MI with proximal LAD occlusion
Image:Ami0003.jpg|Acute MI with proximal LAD occlusion
Image:Ami0013.jpg|Acute MI with LAD occlusion
Image:Ami0013.jpg|Large acute MI with LAD occlusion
Image:Ami0009.jpg|Acute MI with LAD occlusion
Image:ECG_VWI_2wk.jpg|A 2 weeks old anterior infarction with Q waves in V2-V4 and persisting ST elevation, a sign of formation of a [[Cardiac_Aneurysm|cardiac aneurysm]].
Image:ECG_VWI_2wk.jpg|A 2 weeks old anterior infarction with Q waves in V2-V4 and persisting ST elevation, a sign of formation of a [[Cardiac_Aneurysm|cardiac aneurysm]].
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Revision as of 20:48, 22 July 2007

This is part of: Myocardial Infarction

ECG-characteristics:[1]

ST-elevation in leads V1-V6, I and aVL. Maximum elevation in V3, maximal depression in III
later: pathological Q-wave in the precordial leads V2 to V4-V5.
Anterolateral infarct caused by occlusion of the LAD.

Encomprises the anterior part of the heart and a part of the ventricular septum. Is supplied by blood by the LAD.

Examples

References

  1. Wung SF and Kahn DY. A quantitative evaluation of ST-segment changes on the 18-lead electrocardiogram during acute coronary occlusions. J Electrocardiol. 2006 Jul;39(3):275-81. DOI:10.1016/j.jelectrocard.2005.10.007 | PubMed ID:16777513 | HubMed [Wung]