Electrolyte Disorders

Author(s)	J.S.S.G. de Jong
Moderator	T.T. Keller
Supervisor
some notes about authorship

Hyperkalemia

ECG characteristics of hyperkalemia, high blood potassium:

P-waves are widened and of low amplitude due to slowing of conduction
QRS complex:
- QRS widening
- fusion of QRS-T
- loss of the ST segment
Tall tented T waves

The initial part of the QRS complex is often spared as purkinje fibers are less sensitive to hyperkalemia. These changes can also occur in acidosis (via the same mechanism) and during Class IC anti-arrhythmic intoxication.

At concentrations > 7.5 mmol/L atrial and ventricular fibrillation can occur.

Consecutive ECGs of a patient with hyperkalemia. ECG1
Consecutive ECGs of a patient with hyperkalemia. ECG2
Consecutive ECGs of a patient with hyperkalemia. After correction of potassium levels. ECG3
Another patient, potassium of 9.4 mmol/L
Potassium 7.5 mmol/L. More ECGs

Hypokalemia

Hypokalemia, low blood potassium, results in:

ST depression and flattening of the T wave
Negative T waves
A U-wave may be visible

Hypkalemia.jpg

A patient with hypokalemia, prominent QT prolongation. Not the extrasystoles originating from the prolonged T/U wave. This patient definitely needs rhythm monitoring
patient A
patient A
patient B
Patient C, Potassiumlevel of 1.5

Hypercalcemia

Hypercalcemia, high blood calcium, speeds repolarization. Characteristics of hypercalcemia:

Mild: broad based tall peaking T waves
Severe: extremely wide QRS, low R wave, disappearance of p waves, tall peaking T waves.

A patient with severe hypercalcemia: Calcium 4.6 mmol/L, albumin 37 g/L

Hypocalcemia

ECG-characteristics of hypocalcemia, low blood calcium:

Narrowing of the QRS complex
Reduced PR interval
T wave flattening and inversion
Prolongation of the QT-interval
Prominent U-wave
Prolonged ST and ST-depression