191
edits
Chamber Hypertrophy and Enlargment: Difference between revisions
Jump to navigation
Jump to search
Chamber Hypertrophy and Enlargment (view source)
Revision as of 22:30, 18 September 2007
, 18 September 2007no edit summary
No edit summary |
|||
| Line 1: | Line 1: | ||
{{authors| | |||
|mainauthor= [[user:Drj|J.S.S.G. de Jong]] | |||
|moderator= [[user:Drj|J.S.S.G. de jong]] | |||
|supervisor= | |||
}} | |||
In hypertrophy the heart muscle is thicker. This can have different causes. Left ventricular hypertrophy results from an increase in left ventricular workload, e.g. during hypertension or aortic valve stenosis. Right ventricular hypertrophy results from an increase in right ventricular workoad, e.g. emphysema or pulmonary embolisation. | In hypertrophy the heart muscle is thicker. This can have different causes. Left ventricular hypertrophy results from an increase in left ventricular workload, e.g. during hypertension or aortic valve stenosis. Right ventricular hypertrophy results from an increase in right ventricular workoad, e.g. emphysema or pulmonary embolisation. | ||
These causes are fundamentally different from [[Miscellaneous#Hypertrophic_Obstructive_Cardiomyopathy|hypertrophic obstructive cardiomyopathy (HCM)]], which is a congenital misallignment of cardiomyocytes resulting in hypertrophy. | These causes are fundamentally different from [[Miscellaneous#Hypertrophic_Obstructive_Cardiomyopathy|hypertrophic obstructive cardiomyopathy (HCM)]], which is a congenital misallignment of cardiomyocytes resulting in hypertrophy. | ||