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Repolarization (ST-T,U) Abnormalities: Difference between revisions
Repolarization (ST-T,U) Abnormalities (view source)
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Repolarization can be influenced by many factors, including electrolyte shifts, ischemia, structural heart disease (cardiomyopathy) and (recent) arrhythmias. Although T/U wave abnormalities are rarely specific for one disease, it can be useful to know which conditions can change repolarization. | |||
*Early repolarization (normal variant)[[File:early_repol.svg|thumb|300px]] | |||
*Juvenile T waves (normal variant) | |||
*Nonspecific abnormality, ST segment and/or T wave | |||
*ST and/or T wave suggests ischemia | |||
*ST suggests injury | |||
*ST suggests ventricular aneurysm | |||
*Q-T interval prolonged | |||
*Prominent U waves | |||
*Cardiac Memory|Cardiac Memory | |||
'''Early repolarization''' is a normal variant of the ST segment, seen in 2-5% of patients, especially young men. Early repolarization is characterized by elevation of the J point and the beginning of the ST segment as well as elevation of the ST segment itself<cite>Wellens</cite>. The ST segment may be concave up (cup-like) or concave (dome-like). These findings are most often present in the middle chest leads V2-V5. | |||
Recently a different form of early repolarization has been associated with idiopathic ventricular fibrillation. This form is most often seen in lead II and consists of a 'hump' in the tail of the QRS complex, without ST elevation. | |||
==References== | |||
<biblio> | |||
#Wellens pmid=18463384 | |||
#Tikkanen pmid=19917913 | |||
</biblio> | |||