Nightly phenomena, day time work: Difference between revisions

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m (New page: R. Tukkie, R. Rienks, A.A.M. Wilde In 2002, an a trial demand inhibited (AAI) pacemaker was implanted in a young male (born 1984) because of a primary arrhythmia disorder. On a routine H...)
 
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'''What is the consequence of the observed phenomenon in question 1 in the setting of this patient’s primary electrical disease?'''
'''What is the consequence of the observed phenomenon in question 1 in the setting of this patient’s primary electrical disease?'''


As a result of the pause after the non-conducted atrial paced beat, the QT interval further prolongs significantly to QTc 740 ms, increasing the risk of torsades de pointes. QT prolongation at bradycardia is a feature of
As a result of the pause after the non-conducted atrial paced beat, the QT interval further prolongs significantly to QTc 740 ms, increasing the risk of torsades de pointes. QT prolongation at bradycardia is a feature of type III LQTS.  
 
type III LQTS.  


'''What action should be undertaken?'''
'''What action should be undertaken?'''


It is mandatory to prevent relative bradycardia and pauses due to Wenckebach block in this patient. This can be achieved by adding a ventricular lead and programming the new pacemaker to duel-chamber with an AV delay long enough to advocate intrinsic AV conduction to the ventricle. Our patient underwent an uneventful upgrade and a control Holter recording showed excellent prevention of pauses.
It is mandatory to prevent relative bradycardia and pauses due to Wenckebach block in this patient. This can be achieved by adding a ventricular lead and programming the new pacemaker to duel-chamber with an AV delay long enough to advocate intrinsic AV conduction to the ventricle. Our patient underwent an uneventful upgrade and a control Holter recording showed excellent prevention of pauses.