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Ectopic Complexes: Difference between revisions
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{{authors| | |||
|mainauthor= [[user:Drj|J.S.S.G. de Jong]] | |||
|moderator= [[user:Drj|J.S.S.G. de jong]] | |||
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}} | |||
The pacemakercells in the sinusnode are not the only cells in the heart that can depolarize spontaneously. Actually all cardiomyoctyes have this capacity. The only reason why the sinusnode 'rules' is that it is the fastest pacemaker of the heart. From sinusnode to ventricle all healthy cardiomyocytes can function as a ectopic pacemaker. Ectopic pacemaker activity can originate from the atria (60-80 bpm), AV-node (40-60 bpm) and the ventricles (20-40 bpm). So, as the sinus rate drops (e.g. during atrial infarction), other cells can take over. The configuration of ectopic beats or extrasystoles, as seen on the ECG, reveals its origin, whether they are [[#Atrial premature beats|atrial]], nodal or [[#Premature ventricular beats(PVB) / Venticular extrasystoles (VES)|ventrical]]. | The pacemakercells in the sinusnode are not the only cells in the heart that can depolarize spontaneously. Actually all cardiomyoctyes have this capacity. The only reason why the sinusnode 'rules' is that it is the fastest pacemaker of the heart. From sinusnode to ventricle all healthy cardiomyocytes can function as a ectopic pacemaker. Ectopic pacemaker activity can originate from the atria (60-80 bpm), AV-node (40-60 bpm) and the ventricles (20-40 bpm). So, as the sinus rate drops (e.g. during atrial infarction), other cells can take over. The configuration of ectopic beats or extrasystoles, as seen on the ECG, reveals its origin, whether they are [[#Atrial premature beats|atrial]], nodal or [[#Premature ventricular beats(PVB) / Venticular extrasystoles (VES)|ventrical]]. | ||